Wikipedia

核内受容体コアクチベーター7

核内受容体コアクチベーター7（NCOA7: nuclear receptor coactivator 7）は、ヒトのNCOA7遺伝子によってコードされるタンパク質である[5] [6]。

NCOA7
識別子
記号NCOA7, ERAP140, ESNA1, NCOA7-AS, Nbla00052, Nbla10993, TLDC4, dJ187J11.3, nuclear receptor coactivator 7
外部IDOMIM: 609752 MGI: 2444847 HomoloGene: 65245 GeneCards: NCOA7
遺伝子の位置 (ヒト)
6番染色体 (ヒト)
染色体6番染色体 (ヒト)[1]
6番染色体 (ヒト)
NCOA7遺伝子の位置
NCOA7遺伝子の位置
バンドデータ無し開始点125,781,161 bp[1]
終点125,932,034 bp[1]
遺伝子の位置 (マウス)
10番染色体 (マウス)
染色体10番染色体 (マウス)[2]
10番染色体 (マウス)
NCOA7遺伝子の位置
NCOA7遺伝子の位置
バンドデータ無し開始点30,504,995 bp[2]
終点30,679,322 bp[2]
RNA発現パターン




さらなる参照発現データ
遺伝子オントロジー
分子機能• 血漿タンパク結合
• nuclear receptor coactivator activity
細胞の構成要素• 細胞内
• 細胞核
生物学的プロセス• regulation of transcription, DNA-templated
• transcription, DNA-templated
• positive regulation of transcription by RNA polymerase II
• negative regulation of cellular response to oxidative stress
• negative regulation of peptidyl-cysteine S-nitrosylation
• negative regulation of oxidative stress-induced neuron death
出典:Amigo / QuickGO
オルソログ
種ヒトマウス
Entrez

135112

211329

Ensembl

ENSG00000111912

ENSMUSG00000039697

UniProt

Q8NI08

Q6DFV7

RefSeq
(mRNA)
NM_001122842
NM_001199619
NM_001199620
NM_001199621
NM_001199622

NM_181782

NM_001111267
NM_172495
NM_001358841
NM_001358842

RefSeq
(タンパク質)
NP_001116314
NP_001186548
NP_001186549
NP_001186550
NP_001186551

NP_861447

NP_001104737
NP_766083
NP_001345770
NP_001345771

場所
(UCSC)		Chr 6: 125.78 – 125.93 MbChr 6: 30.5 – 30.68 Mb
PubMed検索[3][4]
ウィキデータ
閲覧/編集 ヒト閲覧/編集 マウス

脚注 編集

  1. ^ a b c GRCh38: Ensembl release 89: ENSG00000111912 - Ensembl, May 2017
  2. ^ a b c GRCm38: Ensembl release 89: ENSMUSG00000039697 - Ensembl, May 2017
  3. ^ Human PubMed Reference:
  4. ^ Mouse PubMed Reference:
  5. ^ “ERAP140, a Conserved Tissue-Specific Nuclear Receptor Coactivator”. Mol Cell Biol 22 (10): 3358–72. (Apr 2002). doi:10.1128/MCB.22.10.3358-3372.2002. PMC 133794. PMID 11971969. https://www.ncbi.nlm.nih.gov/pmc/articles/PMC133794/. 
  6. ^ “Entrez Gene: NCOA7 nuclear receptor coactivator 7”. 2021年8月16日閲覧。

参考文献 編集

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外部リンク 編集

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